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glutathione and breast cancer risk An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes glutathione S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in
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glutathione and breast cancer risk An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes glutathione S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in
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Association of glutathione S transferase T1, M1, and P1 polymorphisms TCRM Dove Medical Press The potential anti cancer effects of melatonin on breast cancer Glutathione in Cancer Cell Death Frontiers Glutathione responsive and exhausting metal nanomedicines for robust synergistic cancer therapy
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glutathione and breast cancer risk An integrative multi-omics analysis leveraging Mendelian randomization and subsequent experimental validation prioritizes glutathione S-transferase mu 5 (GSTM5) as a genomic stability-related gene and a therapeutic vulnerability to PLK1 inhibition in
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